I'm not sure that I agree with the premise of the article that the virus has any effect whatsoever on the actual bacteria.
I read the autopsy report of a COVID victim and here is a basic overview (if you are interested). Note: the picture is from a horse but the function is the same.
The small sacs in your lungs where oxygen and CO2 is exchanged look a lot like this. If you can see, there are two types of cells called pneumocytes. The Type I pneumocytes is where gas exchange occurs--they are very, very thin and gas diffuses across the cell into the blood and vice versa. The Type 2 pneumocytes produce what is called surfactant and, in recovery, differentiate into new type 1 pneumocytes. Surfactant helps keep the alveoli open whenever you exhale. Its like surface tension of water. When babies are born too early these Type 2 pneumocytes are not working well and there is no surfactant. Essentially every alveoli collapses with each breath. Just like when you are blowing up a balloon that first bit is much, much harder to inflate than it is after you have gotten the balloon halfway inflated. Surfactant keeps the alveoli halfway inflated.
This virus attacks these pneumocytes. It damages the Type 1 cells causing them to thicken, swell, and eventually die. The Type 2 cells eventually begin to die as well. As you can imagine these diseased Type 1 cells make it harder to diffuse oxygen. Without type 2 cells your alveoli collapse and it becomes very hard to breath. Physiologically it turns your lungs into those of a premature baby. We treat you the same--steroids, ventilation with positive pressure (keep those alveoli open mechanically!) and high dose oxygen (steeper diffusion gradient).
That is not ALL that this virus does, but it is the mechanism that is most likely to cause death.
That said, get your pneumonia shot. That mess of dead cells, collapsed alveoli is PRIME breeding ground for bacteria that cause pneumonia.