CORONA Why there is some hype on Corona Virus

Knoxville's Joker

Has No Life - Lives on TB
The brief summary is that it appears that the 2017 influenza season never stopped. The physical reporting period for the CDC starts and stops in October along with federal agency guidelines. so 2017 ends October 2018, 2018 ends October 2019, 2019 ends October 2020. Have I confused you yet?

I prefer to deal in fact instead of fiction. To say that we are not being fed tailor made information is an understatement. After having an uncle pass in July 2019 from flu like symptoms and then have another relative diagnosed with covid 19 I am starting to wonder if things are being misrepresented and obfuscated to hide key facts.

Given the passage of months and years is it possible we are already on round 2 or 3 and that is why folks are raising concern?

Let us start with some brief overview courtesy of the CDC:
Human Coronavirus Types
Coronaviruses are named for the crown-like spikes on their surface. There are four main sub-groupings of coronaviruses, known as alpha, beta, gamma, and delta.
Human coronaviruses were first identified in the mid-1960s. The seven coronaviruses that can infect people are:
Common human coronaviruses
1)229E (alpha coronavirus)
2)NL63 (alpha coronavirus)
3)OC43 (beta coronavirus)
4)HKU1 (beta coronavirus)
Other human coronaviruses
5)MERS-CoV (the beta coronavirus that causes Middle East Respiratory Syndrome, or MERS)
6)SARS-CoV (the beta coronavirus that causes severe acute respiratory syndrome, or SARS)
7)SARS-CoV-2 (the novel coronavirus that causes coronavirus disease 2019, or COVID-19) Coronavirus Disease 2019 (COVID-19)
People around the world commonly get infected with human coronaviruses 229E, NL63, OC43, and HKU1.
Sometimes coronaviruses that infect animals can evolve and make people sick and become a new human coronavirus. Three recent examples of this are 2019-nCoV, SARS-CoV, and MERS-CoV.



Estimated Influenza Illnesses, Medical visits, Hospitalizations, and Deaths in the United States — 2017–2018 influenza season
All ages 2017 flu season:
Symptomatic Illnesses 44,802,629
Medical Visits 20,731,323
Hospitalizations 808,129
Deaths 61,099
Death concentrations:
50-64 6751 death, 65+ 50903 deaths

all ages 2018 flu season:
Symptomatic Illnesses 35,520,883
Medical Visits 16,520,350
Hospitalizations 490,561
Deaths 34,157

2019-2020 U.S. Flu Season: Preliminary Burden Estimates
CDC estimates* that, from October 1, 2019, through March 14, 2020, there have been:
38,000,000 – 54,000,000flu illnesses
17,000,000 – 25,000,000flu medical visits
390,000 – 710,000flu hospitalizations
23,000 – 59,000flu deaths
*Because influenza surveillance does not capture all cases of flu that occur in the U.S., CDC provides these estimated ranges to better reflect the larger burden of influenza. These estimates are calculated based on CDC’s weekly influenza surveillance data and are preliminary.
 
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Knoxville's Joker

Has No Life - Lives on TB
The 2018-2019 flu season may not have been as severe as the one that came before it, but it set a record of its own, the Centers for Disease Control and Prevention (CDC) say. It was the longest in a decade, lasting 21 weeks.
Fewer illnesses, hospitalizations and deaths were reported this year than during last year’s notoriously brutal flu season, earning the 2018-2019 season an overall severity rating of “moderate,” according to a new CDC recap. But the length and trajectory of the most recent flu season—which began in November, peaked in mid-February and trailed off in April—was unique, the CDC says.
Most flu seasons start off with lots of infections from influenza A viruses, which can be more severe and less responsive to vaccination than other subtypes, while generally less-severe influenza B viruses often strike later. But this year, the CDC says, two different phases of influenza A activity dominated the season, contributing to its unusual length. H1N1 circulated widely from October to mid-February, then H3N2 picked up from mid-February into the spring, according to the new report.
Even still, high early-season vaccination rates and a relatively effective annual vaccine appeared to help suppress illnesses. In total, the CDC estimates that up to 42.9 million people got sick during the 2018-2019 flu season, 647,000 people were hospitalized and 61,200 died. That’s fairly on par with a typical season, and well below the CDC’s 2017-2018 estimates of 48.8 million illnesses, 959,000 hospitalizations and 79,400 deaths.
Pediatric hospitalizations were similar to last year’s levels, the CDC says, but there were fewer pediatric deaths: 116 children died from the flu this year, compared to 183 last year.
Although the 2018-2019 flu season is over, the CDC is already reminding people to get vaccinated ahead of the 2019-2020 season, since it’s the best way to reduce the risk of getting and transmitting influenza. October, ahead of the bulk of flu season, is the best time to get vaccinated, according to the CDC.
 

Knoxville's Joker

Has No Life - Lives on TB
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Irrefutable: The coronavirus was engineered by scientists in a lab using well documented genetic engineering vectors that leave behind a “fingerprint”
Submitted by Dave Hodges on Tuesday, February 4, 2020 - 11:17.

By Mike Adams
Newstargat.com

Every virology lab in the world that has run a genomic analysis of the coronavirus now knows that the coronavirus was engineered by human scientists. The proof is in the virus itself: The tools for genetic insertion are still present as remnants in the genetic code. Since these unique gene sequences don’t occur by random chance, they’re proof that this virus was engineered by scientists in a lab.
But the WHO and CDC are covering up this inconvenient fact in order to protect communist China and its biological weapons program, since no government wants the public to know the full truth about how frequently government-run labs experience outbreaks. Decades ago, for example, the U.S. Army ran an Ebola bioweapons lab in the United States, where a monkey infected one of the scientists there. The strain turned out to be infectious only in monkeys, not humans, so the world dodged a bullet, but the U.S. Army “nuked” the entire facility with chemical bombs, killing all the monkeys and wiping out any last remnant of the virus on U.S. soil.
You can read the full details of that incident in the book The Hot Zone by Richard Preston. We’ve also covered it at NaturalNews.com, where this book description is reprinted:
In 1989, Reston, VA — one of the most famous U.S. planned communities located about 10 miles from Washington DC — stood at the epicenter of a potential biological disaster. This well-known story was narrated by Richard Preston in a bone chilling account related to the recognition and containment of a devastating tropical filovirus at a monkey facility — the Reston Primate Quarantine Unit.

That outbreak occurred because Ebola was found to be spreading through the air ducts, confirming that Ebola can spread through the air. This simple fact was vigorously covered up by the entire medical establishment during the Ebola scare in the United States many years later, where the CDC transported an infected patient to a hospital in Dallas, subsequently infecting a nurse who was treated with highly toxic chemicals that caused permanent kidney damage (she later sued the hospital for the damage she suffered).
The reason this is relevant is because in order to understand the coronavirus situation in China, we must first realize that virology research labs routinely experience lapses in containment. Even the United States has failed to contain deadly viral strains when trying to study them. China’s BSL-4 labs have experienced multiple accidental releases of SARS strains, and this new coronavirus is now confirmed to be an engineered strain that was either used in bioweapons research or vaccine experiments.
The genomic coding in the virus is not natural, in other words. Just as you would never encounter a snake in the desert that’s writing a book containing words and grammatical structure, the genetic sequences now identified in the coronavirus strain are, without question, proof that human engineers have been tinkering with the strain.
How to genetically engineer viruses: the pShuttle vector
One of the tools used to accomplish this genetic engineering is called pShuttle. It’s a genetic tool set that can carry a payload of genes to be inserted into the target virus.
Researchers engaged in genetic engineering can purchase the pShuttle sequence from online retailers such as AddGenes.org, which sells the sequence for $75, shipped in “bacteria as agar stab.”
The following map outlines the complete gene sequence of the pShuttle tool:
The method for using pShuttle is described in a PubMed document entitled, “A simplified system for generating recombinant adenoviruses.”
The summary of the paper describes, “a strategy that simplifies the generation and production of such viruses.” Here’s how the process works to achieve genetic engineering of viruses:
A recombinant adenoviral plasmid is generated with a minimum of enzymatic manipulations, using homologous recombination in bacteria rather than in eukaryotic cells. After transfections of such plasmids into a mammalian packaging cell line, viral production is conveniently followed with the aid of green fluorescent protein, encoded by a gene incorporated into the viral backbone. Homogeneous viruses can be obtained from this procedure without plaque purification.
The paper describes how this approach will, “expedite the process of generating and testing recombinant adenoviruses.”
During this process, of course, the pShuttle leaves behind unique code, a “fingerprint” of the genetic modification. It is this fingerprint that has now been identified in the coronavirus.
As revealed by genomics researcher James Lyons-Weiler in this bombshell analysis article, the pShuttle genetic code is found in the coronavirus that’s circulating in the wild.
This is proof that the virus has been engineered by human scientists.
“IPAK researchers found a sequence similarity between a pShuttle-SN recombination vector sequence and INS1378,” writes Lyons-Weiler for IPAK:
Another gene sequence also shows a 92% match with the Spike protein from the SARS coronavirus:
The process for achieving this was patented by Chinese researchers as shown in this patent link.
The pShuttle vector was used to insert SARS genes into the coronavirus, a process that makes it deadly to humans. “The very researchers conducting studies on SARS vaccines have cautioned repeatedly against human trials,” warns Lyons-Weiler:
The disease progression in of 2019-nCoV is consistent with those seen in animals and humans vaccinated against SARS and then challenged with re-infection. Thus, the hypothesis that 2019-nCoV is an experimental vaccine type must be seriously considered.
He also warns about, “studies that have reported serious immunopathology in animals – rats, ferrets, and monkeys – in which animals vaccinated against coronoviruses tended to have extremely high rates of respiratory failure upon subsequent exposure in the study when challenged with the wild-type coronavirus.”
He concludes:
If the Chinese government has been conducting human trials against SARS. MERS, or other coronviruses using recombined viruses, they may have made their citizens far more susceptible to acute respiratory distress syndrome upon infection with 2019-nCoV coronavirus.
Brighteon.com/a4d2afed-56c6-4602-b6ab-6f777ba4a69a

Another doctor from Beijing Medical University warns the virus appears to be genetically engineered
Lyons-Weiler is not alone in his assessment of the genetic engineering origins of the coronavirus. Dr. Yuhong Dong, who holds a doctorate degree in infectious diseases from Beijing University, writes in The Epoch Times:
Based on recently published scientific papers, this new coronavirus has unprecedented virologic features that suggest genetic engineering may have been involved in its creation. The virus presents with severe clinical features, thus it poses a huge threat to humans. It is imperative for scientists, physicians, and people all over the world, including governments and public health authorities, to make every effort to investigate this mysterious and suspicious virus in order to elucidate its origin and to protect the ultimate future of the human race.
Dr. Yuhong reminds us that a Jan. 30 science paper published in The Lancet concludes that, “recombination is probably not the reason for emergence of this virus.” In other words, this did not occur through natural mutations in the wild.
He also points to a Jan. 27th study by five Greek scientists who also concluded the coronavirus has no lineage to other viruses in the “family tree” that’s found in the wild. He writes:
A Jan. 27 2020, study by 5 Greek scientists analyzed the genetic relationships of 2019-nCoV and found that “the new coronavirus provides a new lineage for almost half of its genome, with no close genetic relationships to other viruses within the subgenus of sarbecovirus,” and has an unusual middle segment never seen before in any coronavirus. All this indicates that 2019-nCoV is a brand-new type of coronavirus. The study’s authors rejected the original hypothesis that 2019-nCoV originated from random natural mutations between different coronaviruses.
“No bats were sold or found at the Huanan seafood market”
Dr. Yuhong writes about The Lancet study by authors Roujian Lu et al., from the China Key Laboratory of Biosafety, National Institute for Viral Disease Control and Prevention, Chinese Center for Disease Control and Prevention, repeating a quote from that paper:
First, the outbreak was first reported in late December 2019, when most bat species in Wuhan are hibernating. Second, no bats were sold or found at the Huanan seafood market, whereas various non-aquatic animals (including mammals) were available for purchase. Third, the sequence identity between 2019-nCoV and its close relatives bat-SL-CoVZC45 and bat-SL-CoVZXC21 was less than 90%. Hence, bat-SL-CoVZC45 and bat-SL-CoVZXC21 are not direct ancestors of 2019-nCoV.
In other words, it isn’t from bats.
That means the entire mainstream media is lying to us about the real origins of the coronavirus.
That same paper goes on to underscore the misinformation in the official explanation, stating, “Many of the initially confirmed 2019-nCoV cases—27 of the first 41 in one report, 26 of 47 in another—were connected to the Wuhan market, but up to 45%, including the earliest handful, were not. This raises the possibility that the initial jump into people happened elsewhere.”
Both Lu (in The Lancet paper linked above) and Lyons-Weiler point to the presence of a SARS binding protein sequence in the coronavirus that allows it to easily infect human cells. As explained in The Epoch Times:
…despite considerable genetics distance between the Wuhan CoV and the human-infecting SARS-CoV, and the overall low homology of the Wuhan CoV S-protein to that of SARS-CoV, the Wuhan CoV S-protein had several patches of sequences in the receptor binding (RBD) domain with a high homology to that of SARS-CoV. The residues at positions 442, 472, 479, 487, and 491 in SARS-CoV S-protein were reported to be at receptor complex interface and considered critical for cross species and human-to-human transmission of SARS-CoV. So to our surprise, despite replacing four out of five important interface amino acid residues, the Wuhan CoV S-protein was found to have a significant binding affinity to human ACE2. …The Wuhan CoV S-protein and SARS-CoV S-protein shared an almost identical 3-D structure in the RBD domain, thus maintaining similar van der Waals and electrostatic properties in the interaction interface. Thus the Wuhan CoV is still able to pose a significant public health risk for human transmission via the S protein–ACE2 binding pathway. (emphasis added)
As Dr. Yuhong asks, “How could this novel virus be so intelligent as to mutate precisely at selected sites while preserving its binding affinity to the human ACE2 receptor? How did the virus change just four amino acids of the S-protein? Did the virus know how to use Clustered Regularly Interspaced Short Palindromic Repeats (CRISPR) to make sure this would happen?”
It couldn’t happen by chance, in other words. The coronavirus is not a random mutation in the wild. It was engineered.
Many other scientists around the world are now investigating the gene sequences found in the coronavirus, and they are increasingly concluding that elements of the virus have been engineered.
Many of those scientists are being threatened and censored. One paper has so far been forced to be withdrawn and revised, no doubt to remove the key conclusions that point to the genetic engineering origins of the coronavirus, but the proof of its engineering cannot be denied forever.
Brighteon.com/0bddc3da-f590-445e-ac38-9a74e358a57c

Either the coronavirus was genetically engineered, or the science establishment is going to have to throw out the entire field of genomics research and claim it isn’t real
Eventually, the science establishment is either going to have to conclude that this coronavirus strain was engineered, or that all the laws of genetics science don’t work, and gene sequencing is imaginary (sort of like transgenderism by the “progressive” Left, which has already abandoned biological reality).
So far they’ve tried to bamboozle the public into believing this is all some sort of accident from Mother Nature, but that has only worked because most of the public doesn’t understand enough science to counter the official propaganda. However, there are more than enough independent scientists around the world to prove that this pandemic strain was engineered by humans. More evidence is coming out each day.
Interestingly, as this article is going to press, all the official numbers of infections and deaths from coronavirus have been frozen for about 14 hours and counting, almost as if every nation of the world has agreed to stop reporting new numbers. This may be a temporary situation that gets resolved in the next few hours, but it’s highly suspicious. For the last week, we’ve been getting new updates about every 12 hours or sooner, and we’ve never seen the count frozen for this long.
At the same time, an 11th case of coronavirus has now been confirmed by the CDC in the United States, revealing that infections are continuing to spread in the USA, despite the efforts of the CDC to contain the outbreak.
 

Knoxville's Joker

Has No Life - Lives on TB
Tedros Adhanom Ghebreyesus, director general of the World Health Organization, has said that ‘Africa should wake up’ to the threat of coronavirus by ramping up testing. Photograph: Fabrice Coffrini/AFP via Getty Images
The world’s most vulnerable people could be last in line for support to deal with the coronavirus outbreak, experts have warned.
Countries already dealing with humanitarian and refugee crises face a struggle to find the resources to deal with the pandemic by the time it reaches them, specialists said in a webinar hosted by the New Humanitarian news agency on Thursday.
Protective equipment and resources for testing are already a concern for China and Europe, but a third wave could leave developing countries with weakened health systems in a worse position – even though most currently have a relatively small number of cases.
Experts working with refugees, on humanitarian responses and in global health warned that the international community needs to begin working closely with governments around the world to help the most vulnerable.
“I don’t think we’re quite ready yet for the fights that are coming over limited supplies of personal protective equipment, limited supplies of vaccines and limited supplies of therapeutics,” said Jeremy Konyndyk, senior policy fellow at the Center for Global Development in Washington DC.
“When the first 100m doses of the vaccines come out, it’s going to be a big fight over who gets those, and it’s going to be very important that [they don’t] just go to those who can afford them.”
Africa has had about 600 cases, compared with thousands in many individual European countries. But the World Health Organization chief, Tedros Adhanom Ghebreyesus, said more testing was needed across the continent.

“Africa should wake up, my continent should wake up,” said Tedros.
But with testing limited in much of the world, including the UK, developing countries faced a major challenge, said Karl Blanchet, director of the Geneva-based Centre for Education and Research in Humanitarian Action.
“Testing has to happen, it is the priority. The problem is low-income countries are probably arriving on the third wave, after China, after Europe. Access to tests is going to be problematic,” said Blanchet.
While many richer countries were currently focused on their own populations and economies, the humanitarian community would have to look at changing the way it works, said the experts, by investing in the strengthening of worldwide public health systems rather than focusing on single-issue campaigns.
“This is not just about providing services in a refugee site or a conflict-affected area, this is really about the fundamental of health systems and about health-seeking and health-protecting behaviour across entire countries,” said Konyndyk. “That is not something that we in the humanitarian sector are always terribly well set up for.”
Konyndyk said the sector would have to work more with governments and local organisations, who have the trust of the communities with which they work.
Virginie Lefèvre, programme and partnerships coordinator with the Lebanese NGO Amel Association, said she was already seeing examples of local workers leading the way.
“The locally-led response, the community-based initiatives are working despite the fear. All those workers are afraid – as we can understand, because they have been exposed,” she said.
Lefèvre praised such workers for quickly setting up mobile clinics and using technology to communicate with people.
“We have good locally-led responses, but it needs to be better coordinated with other actors in-country and at the international level,” she added.
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